Search the Community
Showing results for tags 'biotin'.
Found 3 results
I am 23 and last year my mother noticed the hair near my crown thinning. I have been pretty paranoid since. I get a lot of conflicting opinions from people close to me, ranging from ‘everything is perfectly’ fine to ‘you will be bald within 2-3 years’. Thought I would get an opinion from more experienced people on here who have gone through similar journeys. I am currently taking daily: 10,000 mcg biotin pills, 500 mg Saw Palmetto pills, 350 mg beta-sitosterole and shampoo daily with either bitoin shampoo/alpecin (both which claim to block DHT, the latter using caffeine). And every 3-4 days I also use Nizoral 2% since I have dandruff. I am a bit afraid to start fina, mainly because of side-effects which 2% of the people noticed. Also I am not sure how much it would cost to get a prescription as I don’t have a lot of money and my insurance will not cover appointment for this kind of hairloss (at least thats what I think) so 2 questions for the community: 1) How bad is the hair loss from pictures (taken from iphone front camera) and how much time approximately before it is extremely noticeable and irreversible? 2) What treatment should I opt for? Shouls I continue my regimen or switch to fina ans minoxdil combo? Anyone noticed positive effects from saw palmetto and/or beta-sitosterole? Thanks in advance!
I have peruse this site for a while now, and though it was best to join so I could interact with the community. If you need to gain access to certain studies I can help with that as well. I wanted to see if anyone has had any experiences with soy or tea and reduced hair loss. --While it does not seem to reduce hair loss, Corvettester's experience with Biotin (http://www.hairrestorationnetwork.com/eve/163314-corvettesters-biotin-experience.html) is encouraging, and I will start taking this. --Personally I have tried saw palmetto for over a year (450mg) and while I saw reduced hair loss and zero side effects, I am going to move on to other treatments. However, I would not discourage its use. One study's (Potency of a Novel Saw Palmetto Ethanol Extract, SPET-085, for Inhibition of 5α-Reductase II) review of the literature indicates that "Clinical studies of SPE have been equivocal, with some showing significant results and others not. These inconsistent results may be due, in part, to varying bioactivities of the SPEs used in the studies." The above study indicated success in reducing the enzyme that converts testosterone to DHT. It is attached! --No relationship was found between Vitamin D levels and alopecia in a 2008 study(Does degree of baldness influence vitamin D status?) --Vitamin E did not help reduce alopecia in one study (High-dose alpha-tocopherol as a preventive of doxorubicin-induced alopecia.) However one cannot rule out the effect of the anti-cancer drug as a confounding factor. --Spearmint seems to help reduce hirsutism in women by reducing free testosterone (Effect of spearmint (Mentha spicata Labiatae) teas on androgen levels in women with hirsutism.) Here is the abstract: Mentha spicata Labiatae, known as spearmint and Mentha piperita Labiatae, known as peppermint can be used for various kinds of illnesses in herbal medicine and flavoring in industry. M. spicata Labiatae grows on the Anamas plateau of Yenithornarbademli town of Isparta, located in southwest part of Turkey. In this town, clinicians thought that consumption of tea steeped with M. spicata or M. piperita caused a diminished libido. Because antiandrogenic effects of spearmint and peppermint were found previously in rats, it was decided to observe the effect of this herbal tea on the androgen levels in hirsute women.Twenty-one female hirsute patients, 12 with polycystic ovary syndrome and 9 with idiopathic hirsutism were included to the study. They were took a cup of herbal tea which was steeped with M. spicata for 5 days twice a day in the follicular phase of their menstrual cycles. After treatment with spearmint teas, there was a significant decrease in free testosterone and increase in luteinizing hormone, follicle-stimulating hormone and estradiol. There were no significant decreases in total testosterone or dehydroepiandrostenedione sulphate levels. Spearmint can be an alternative to antiandrogenic treatment for mild hirsutism. Further studies are needed to test the reliability of these results and the availability of spearmint as a drug for hirsutism. --Soy and tea. The only study I found (Soy phytochemicals and tea bioactive components synergistically inhibit androgen-sensitive human prostate tumors in mice.) indicated success in reducing dht with black but not green tea. I have attached the study. Here are some interesting extracts: Combined effects of soy phytochemicals and tea on serum testosterone and DHT concentrations. Mice treated with black tea tended to have a greater serum testosterone concentration (34.4%, P 0.50) and had a 72% lower DHT concentration than controls (P 0.05), suggesting that black tea may contain components that inhibit the activity of 5- reductase, an enzyme that converts testosterone to the more bioactive DHT. Green tea tended to increase serum testosterone and DHT levels by 73.8% (P 0.14) and 194% (P 0.076), respectively. The combination of SPC (Soy extract) and green tea reduced serum levels of DHT (P 0.05). ... Several in vitro and in vivo studies have shown that green and black tea polyphenols have chemopreventive effects on prostate carcinogenesis (16,18,27). Studies of tea polyphenols suggest that epigallocatechin gallate (EGCG) is the major bioactive component in green tea and less is present in black tea. Black tea also contains other tea polyphenols such as theaﬂavins and thearubigins (28). Multiple studies also suggest that components in dietary soy have anticarcinogenic effects (Fig. 2D), suggesting that black tea may have bioactive components that inhibit the conversion of testosterone to DHT,presumably via inhibition of 5-reductase in this SCID-LNCaP animal model. It is unclear whether black tea theaﬂavins, EGCG and/or other components are responsible for this function in vivo. On the other hand, green tea did not reduce the serum levelof DHT, but instead tended to increase it (P 0.076) (Fig.2D), and we found that green tea treatment did not inhibittumor growth (Fig. 1A). Green tea contained more EGCGthan black tea (Table 1), and studies have shown that EGCGinhibits the activity of 5-reductase (38). These results derived from our animal model suggest that, although EGCGmay be a potent antitumor agent in green tea and inhibit5-reducatase activity, green tea contains other constituentsthat may counteract EGCG’s antitumor activity, in part bycounteracting its modulation of 5-reducatase activity. Further research is required to identify these constituents andstudy their effects and/or their interactions with other components on prostate cancer. Our results demonstrate the importance of evaluating the beneﬁt of whole tea products,rather than just isolated tea catechins or EGCG, on prostatecancer prevention because other tea constituents may playimportant roles. Green tea combined with SPC reduced total testosteroneand DHT levels (Fig. 2A, B), suggesting that interactivemodulation of androgen levels is one of the important mechanisms for the synergistic prevention of prostate cancer progression by the soy/green tea combination. This study supportsthe use of appropriate combinations of bioactive dietaryagents, such as soy and tea, as effective nutritional regimens forprostate cancer prevention and treatment. Has anyone has any experience with soy and/or black tea? J. Nutr.-2003-Zhou-516-21.pdf Potency of a Novel Saw Palmetto Ethanol Extract.pdf
Hi all, I have decided to move on to a FUE HT, I am just looking for the best timing! I was never into meds and I only recently started using minoxidil, biotin and Revivogen shampoo. I cannot go to Finasteride for medical reasons. My hair loss is quite aggressive the last months. This is a stressful period for me too, not sure if this has an impact. I am 31. I am not sure if I should proceed to the HT asap (next 1-2 months) or wait (and hope!) for my hair loss to stabilise (and go for December). I am worried about the shock loss in the first case. Any help here? Many thanks!